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In general, patients had an incremental benefit from PCI for 6 to 24 months; patients with more severe angina had a greater benefit from PCI.

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Other secondary end points included failure of therapy, defined as the composite of cardiovascular death, MI, recurrent ischemia, a positive Holter for ischemia, hospitalization for new or worsening heart failure, or an early positive exercise tolerance test (evidence for ischemia before completing 12 minutes of a modified Bruce protocol or equivalent).

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This report evaluates the current state of evidence regarding effectiveness and harms of noninvasive technologies for the diagnosis of coronary artery disease (CAD) or dysfunction that results in symptoms attributable to myocardial ischemia in stable symptomatic patients who have no known history of CAD.

The increase in adverse events seen with the 1500 mg dose in MARISA is disproportionately larger than the increase in anti-anginal efficacy; thus, the 1500 mg twice-daily dose is not recommended for clinical use.

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Health Status and Quality of Life in Patients With Stable Coronary Artery Disease and Chronic Kidney Disease Treated With Optimal Medical Therapy or Percutaneous Coronary Intervention (Post Hoc Findings from the COURAGE Trial).

Coronary arteriography (also called coronary angiography or cardiac catheterization) uses X-rays to follow dye injected into the heart or the coronary arteries.

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The task of developing ESC Guidelines covers not only the integration of the most recent research, but also the creation of educational tools and implementation programmes for the recommendations.

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Men with angina were found to have an increased risk of subsequent acute myocardial infarction and coronary heart disease related death than women.

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Most patients were taking statins (82 percent) and angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (88 percent).

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Older adults may be more sensitive to the side effects of this drug, especially QT prolongation (see above).

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Although the FDA medical officer in charge of reviewing ranolazine recommended that ranolazine's professional product labeling display a black box warning about potential disruption in the heart's electrical cycle, the drug does not have a black box warning.

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DM 35 to 64 years of age have cardiovascular disease and that this proportion is as high as 45% among DM patients 65 to 74 years of age.

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Long-term follow-up studies involving patients receiving ICDs for primary prevention in this setting are needed to inform decision making.

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In still other embodiment, the two active ingredients are co-formulated into a combined dosage unit.

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The Guidelines emphasize the importance of OMT in all patients and for both procedures, and the pivotal role of the heart team in most decisions over revascularization in patient with multivessel or left main disease.

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As such, PChP or Syndrome X is considered in the art to be separate and apart from anginal symptoms which are due to obstructive coronary artery disease.

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Study MDRD Modification of Diet in Renal Disease MERLIN Metabolic Efficiency with Ranolazine for Less Ischaemia in Non-ST-Elevation Acute Coronary Syndromes MERLIN-TIMI 36 Metabolic Efficiency with Ranolazine for Less Ischemia in Non-ST-Elevation Acute Coronary Syndromes: Thrombolysis In Myocardial Infarction MET metabolic equivalents MI myocardial infarction MICRO-HOPE Microalbuminuria, cardiovascular and renal sub-study of the Heart Outcomes Prevention Evaluation study MPI myocardial perfusion imaging MRI magnetic resonance imaging NO nitric oxide NSAIDs non-steroidal anti-inflammatory drugs NSTE-ACS non-ST-elevation acute coronary syndrome NYHA New York Heart Association OAT Occluded Artery Trial OCT optical coherence tomography OMT optimal medical therapy PAR-1 protease activated receptor type 1 PCI percutaneous coronary intervention PDE5 phosphodiesterase type 5 PES paclitaxel-eluting stents PET positron emission tomography PRECOMBAT Premier of Randomized Comparison of Bypass Surgery vs.

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These changes to the underlying myocardial substrate may be complementary to ranolazine's suppression of triggered activity due to early after-depolarizations.

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This new explanatory analysis explicitly examines the stages of exercise and the associated ST-segment deviations on the different regimens of ranolazine.

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Nonetheless, regardless of the conditions of energisation of the mitochondrial inner membrane, the IC 50 for inhibition of mitochondrial complex I by ranolazine was much higher than its therapeutic concentrations.

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